Seasonal Affective Disorder: Giving Meaning to the Holiday Blues

×
Author photo

The Pure TheraPro Team

The Pure TheraPro Education Team is comprised of researchers from diverse backgrounds including nutrition, functional medicine, fitness, supplement formulation & food science. All articles have been reviewed for content, accuracy, and compliance by a holistic integrative nutritionist certified by an accredited institution.
Published
Last updated for accuracy

‘Tis the season to be jolly, right? Perhaps not for everyone. Seasonal Affective Disorder (SAD) is a form of depression characterized by recurrent episodes of major depression, typically occurring smack dab in the middle of the holiday season. While the exact reasons are cause for debate, a consistent association exists between decreased sunlight exposure and the onset of symptoms.

According to the American Psychiatric Association, approximately 5% of the U.S. population experiences SAD, with prevalence rates varying by geographic location. SAD manifests with a distinct pattern, with symptoms dwindling during the spring and summer months, only to make a comeback in the subsequent fall and winter. The demographic distribution of SAD reveals a higher prevalence among women than men, with the onset typically observed in early adulthood. The reduced exposure to natural light during fall and winter contributes to disruptions in circadian rhythms and alterations in serotonin and melatonin levels, potentially aggravating depressive symptoms.

Individuals with SAD frequently report challenges in daily functioning, including impaired concentration, fatigue, and changes in sleep and eating patterns.

Causes and Risk Factors

1. Biological Factors:

The primary biological factor associated with SAD is the disruption of circadian rhythms, the body’s internal clock that regulates various physiological processes. Reduced exposure to natural light during fall and winter months can lead to disturbances in circadian rhythms, impacting sleep-wake cycles, hormone production, and mood-regulating neurotransmitters.

Alterations in serotonin and melatonin levels also play a significant role in the pathophysiology of SAD. Reduced sunlight exposure can lead to decreased serotonin, a neurotransmitter linked to mood regulation. Simultaneously, increased melatonin production, associated with darkness and sleep, may contribute to symptoms such as fatigue and lethargy.

2. Environmental Factors:

Geographic location and latitude are additional environmental factors influencing SAD prevalence. Regions with more pronounced seasonal variations and reduced sunlight, especially at higher latitudes, report higher rates of SAD.

In the United States, the prevalence of SAD varies significantly based on geographic location. For instance, only about 1% of the population in Florida, which is closer to the equator, experiences SAD, compared to 9.9% in Alaska, which is located at a much higher latitude. This trend is consistent with the understanding that the farther an area is from the equator, the less sunlight it receives during the fall and winter months, thereby increasing the risk of SAD.

It's important to note that while the prevalence of SAD is higher in northern climates, environmental factors are not the sole determinants of SAD, as individual susceptibility also plays a significant role.

3. Genetic Predisposition:

Genetic factors contribute to an individual’s susceptibility to SAD. Family studies suggest a hereditary component, with a higher likelihood of developing SAD if a first-degree relative has a history of mood disorders. Specific genes related to circadian rhythms and serotonin regulation are areas of ongoing research in understanding the genetic basis of SAD.

 

Symptoms and Diagnosis

Seasonal Affective Disorder presents a spectrum of symptoms mirroring those of major depressive disorder. The unique characteristic of SAD lies in its seasonal pattern, with symptoms typically surfacing in the fall or winter and subsiding in the spring or summer.

  • Persistent Low Mood: Individuals with SAD often experience prolonged periods of sadness or a sense of emptiness.
  • Loss of Interest or Pleasure: Anhedonia, or the diminished ability to find joy or interest in previously enjoyable activities, is a hallmark symptom.
  • Changes in Sleep Patterns: SAD can lead to disruptions in sleep, manifesting as oversleeping (hypersomnia) or difficulty sleeping (insomnia).
  • Weight Changes: Appetite fluctuations may result in weight gain, often due to increased cravings for carbohydrate-rich foods.
  • Fatigue and Low Energy: Individuals with SAD may experience persistent fatigue, even after adequate rest.
  • Difficulty Concentrating: Impaired focus, attention, and decision-making are common cognitive symptoms.
  • Feelings of Hopelessness or Worthlessness: Negative thought patterns, self-doubt, and a pervasive sense of despair can accompany SAD.

SAD can strain interpersonal relationships as individuals grapple with persistent feelings of sadness and fatigue. Withdrawal from social activities, a common response to depressive symptoms, may lead to isolation and strain relationships with family and friends. The seasonal recurrence of these challenges can contribute to a sense of frustration and helplessness among both individuals with SAD and their support networks.

Individuals with SAD are at an increased risk of developing co-occurring mental health conditions. Anxiety disorders and substance use disorders are commonly reported alongside SAD, necessitating a comprehensive approach to assessment and treatment.

Treatment Options
Dealing with Seasonal Affective Disorder means using different approaches to find what works best for each person. While the effectiveness of interventions may vary, a combination of treatments often yields the most favorable outcomes.

1. Light Therapy:

Light therapy, or phototherapy, involves exposure to bright artificial light that mimics natural sunlight. This treatment aims to compensate for reduced sunlight during fall and winter, helping regulate circadian rhythms and neurotransmitter levels. Light therapy is typically administered daily, and the duration and intensity of exposure vary based on individual requirements.

2. Psychotherapy:

Cognitive-behavioral therapy (CBT) has demonstrated efficacy in treating SAD. CBT helps individuals identify and modify negative thought patterns and behaviors associated with depressive symptoms. Psychoeducation, a component of CBT, also equips individuals with coping strategies to manage seasonal mood changes.

3. Lifestyle Changes:

Incorporating lifestyle modifications can complement formal treatments for SAD. Regular physical activity, maintaining a consistent sleep schedule, and adopting a well-balanced diet contribute to overall well-being. Adequate exposure to natural light, even during overcast days, is encouraged.

A well-balanced diet rich in fruits, vegetables, and whole grains provides essential nutrients that support mental health. Nutritional choices can influence neurotransmitter production and function. Omega-3 fatty acids, found in fish and certain nuts, have been associated with mood regulation.

4. Vitamin D Supplementation:

Given the role of sunlight in Vitamin D synthesis, individuals with SAD may benefit from Vitamin D3 supplementation.

Optimal Vitamin D levels (50 ng/mL or higher) have been shown to support neurotransmitter and hormonal health, positive mood, healthy outlook on life, and much more.

Pure TheraPro’s Vegan D3 + K2 Full-Spectrum is a soy-free, gluten-free, alcohol-free & non-GMO liposomal formula made with zero Chinese ingredients. Formulated with vegan liposomal Vitamin D3 along with two essential forms of liposomal Vitamin K2 (MK-4 + MK-7), it is the ultimate Vitamin D3 supplement to support whole body health, including mood.

In addition to mood, Vitamin D3 helps the body absorb Calcium, while Vitamin K2 helps the body ensure that calcium is properly utilized (i.e., that it is ending up in the bones and not in the heart and blood vessels). Vitamin K2 keeps Calcium in bones and teeth and out of soft tissues.

Most Vitamin D3 comes from lanolin which is obtained from filthy sheeps' grease-wool. We utilize pureshine™, a sustainably harvested source of Cholecalciferol (bioidentical D3) derived entirely from lichen (a naturally vegan plant source of Vitamin D3).

Many people take Vitamin D and calcium supplements thinking they're helping their bones, the truth is without the addition of Vitamin K2, such a health regimen could prove dangerous.

5. Mind-Body Interventions:
Mindfulness-based practices, such as meditation and yoga, can be valuable adjuncts in managing SAD symptoms. These practices promote relaxation, reduce stress, and enhance overall mental resilience.

References

American Psychiatric Association. (2013). Diagnostic and Statistical Manual of Mental Disorders (5th ed.). Arlington, VA: American Psychiatric Publishing.

National Institute of Mental Health. (2022). Seasonal Affective Disorder. https://www.nimh.nih.gov/health/topics/seasonal-affective-disorder/index.shtml

Mayo Clinic. (2022). Seasonal affective disorder (SAD). https://www.mayoclinic.org/diseases-conditions/seasonal-affective-disorder/symptoms-causes/syc-20364651

Rosenthal, N. E., et al. (1984). Seasonal affective disorder: a description of the syndrome and preliminary findings with light therapy. Archives of General Psychiatry, 41(1), 72–80. https://jamanetwork.com/journals/jamapsychiatry/article-abstract/493296

Golden, R. N., et al. (2005). The efficacy of light therapy in the treatment of mood disorders: a review and meta-analysis of the evidence. American Journal of Psychiatry, 162(4), 656–662. https://ajp.psychiatryonline.org/doi/10.1176/appi.ajp.162.4.656

Modell, J. G., et al. (2005). Seasonal affective disorder and its prevention by anticipatory treatment with bupropion XL. Biological Psychiatry, 58(8), 658–667. https://www.sciencedirect.com/science/article/pii/S0006322305004874

Lam, R. W., Levitt, A. J., Levitan, R. D.,; Enns, M. W. (2006). The Can-SAD study: a randomized controlled trial of the effectiveness of light therapy and fluoxetine in patients with winter seasonal affective disorder. American Journal of Psychiatry, 163(5), 805–812. https://ajp.psychiatryonline.org/doi/full/10.1176/ajp.2006.163.5.805

Magnusson, A.; Boivin, D. (2003). Seasonal affective disorder: an overview. Chronobiology International, 20(2), 189–207. https://www.tandfonline.com/doi/abs/10.1081/CBI-120018616

Kasper, S., et al. (1989). Light in the treatment of patients with seasonal affective disorder. Biological Psychiatry, 25(7), 951–960. https://www.sciencedirect.com/science/article/pii/0006322389902446

Young, M. A.; Meaden, P. M. (2007). Seasonal affective disorder: prevalence, detection, and diagnosis. CNS Spectrums, 12(1 Suppl 1), 14–21. https://www.cambridge.org/core/journals/cns-spectrums/article/seasonal-affective-disorder-prevalence-detection-and-diagnosis/FAB5E5357DCAE41E4A259A58EC51E93D